Growing pains - 8 months - Page 5

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by Blitzen on 29 June 2014 - 16:06

My first 2 GSD's had pano and I saw a fair number of cases when I was a tech. I studied with the ortho and radiology dept at Penn when I was a tech. This is what I learned about pano. To the best of my knowledge, none of this has been proven to be inaccurate.

1. The only chance of getting any real funding for the study of pano in dogs is if it can be proven that "growing pains" in human kids is an identical condition. There must be a human model.

2. Reseach funding for canine conditions that are not life threatening is not going to happen either. Pano is self limiting and self curing, no dog ever died directly from pano. The cause and treatment of pano is not a big deal to the veterinary world. It's a nuisance disease.

3. Injecting dogs with narrow from effected dogs just to see if those dogs too will become effected is vivasection which most dog lovers hate and refuse to support. Those are the dogs that will die directly from pano - the ones that are infected, studied and then killed or allowed to die from complications.

4. Dogs fed raw also develop pano as do dogs fed high end dog food like Origen.

5. Most of the dogs we saw at the clinic had a first or second degree relative that also suffered from pano - GSD's, Rotties, Bull mastiffs, Irish Wolfhounds made up the majority of puppies with pano and most were from the same breeders, same lines. Most were males 4 to 7 month old and some had effected littermates that we teated at the same time. Not every puppy with pano was big boned or exceptionally large or heavy for breed and age.

6. The more the dog uses his effect limb/s, the longer it's going to take it to have a permanent remission. Creative owners find other ways to keep the dog healthy and happy rather than working it or letting it have as much exercise as the dog wants.

7. It's always best to allow an effect dog to experience a small amout of pain to insure he doesn't overdo it and increase the recovery time. If you don't dope the pup with Tramadol or Rimadly as most vets tell you to and allow it to experience discomfort, then you can probably let that dog choose its own level of exercise

8. The initial drug of choice is usually buffered aspirin like Ascriptin if that is still on the market. Baby aspirin can be used, but a 5, 6 month old large breed dog is going to need 4 at one dosage. Most owners would rather give one Ascriptin.

9. Pano is  usually disgnosed by gripping the effected long bone in both hands and attempt to bend it. The dog will cry out if it's pano. Xrays are not always diagnostic, especially in the early stages.

10. Pano  used to be called "long bone disease" for obvious reason.

Penn was considered the go to vet school for orthopedic problems and still is as far as I know.

 


Prager

by Prager on 29 June 2014 - 17:06

Pioneer Wife: 

Something, a virus, bacteria or some agent, must trigger pano, if healthy dogs will develop it when injected with 'infected' marrow. OR it disables the body's natural defense against it. It seems to strike regardless sometimes of the level of care and diligence by an owner with respect to diet or exercise.

 Hans: , Not necessarily so. The article itself says that :

1/"Another theory is that the recent trend in high-protein dog foods is to blame. 

 2/Since there is a breed predisposition for panosteitis (German shepherd dogs, Golden retrievers, Basset hounds, Doberman pinschers, and Labrador retrievers), this implies a genetic basis.

Thus there are other theories and possible viable  reasons besides infection.

 . 

 prager hans


by Blitzen on 29 June 2014 - 17:06

And the beat goes on..........................


Prager

by Prager on 29 June 2014 - 17:06

 My reference to God? LOL. I have said what in "What in God's name..."? Jeez I am soooooo sooooorry.

Joan   give it a brake.  I posted my scientific sources above. The "  gobbledygook " sources I am using are derived from besides many other from these sources below: 

 

References:

 

  • Abeles V, Harrus S, Angles JM, Shalev G, Aizenberg I, Peres Y, Aroch I. Hypertrophic osteodystrophy in six weimaraner puppies associated with systemic signs. Vet Rec, 145:130-134, 1999.

  • Fred Lanting: Canine hip dysplasia and other orthopedic disorders 

  • Budsberg, SC, Todhunter, RJ, McNamara, PS, Jr. Use of chondromodulating drugs and substances in the prevention and treatment of osteoarthritis in dogs. In: Bonagura, JD, ed: Kirk's Current Veterinary Therapy XIII. Philadelphia: W.B. Saunders Co., 1999, p. 1018.

  • Olsson SE. Pathophysiology, morphology, and clinical signs of osteochondrosis in the dog. In: Bojrab, MJ, ed: Disease Mechanisms in Small Animal Surgery. Philadelphia: Lea & Febiger, 1993, p.777.

  • Schrader SC. Differential diagnosis of nontraumatic causes of lameness in young growing dogs. In: Bonagura, JD, ed: Kirk's Current Veterinary Therapy XII. Philadelphia: W.B. Saunders Co., 1995, p. 1171.

  • Schrader, SC. The use of the laboratory in the diagnosis of joint disorders of dogs and cats. In: Bonagura, JD, ed: Kirk's Current Veterinary Therapy XII. Philadelphia: W.B. Saunders Co., 1995, p. 1166.

  • Schulz KS, Payne JT, Aronson E. Escherichia coli bacteremia associated with hypertrophic osteodystrophy in a dog. J Am Vet Med Assoc., 199:1170-1173, 1991.

  • Schwarz, PD. Canine elbow dysplasia. In: Bonagura, JD, ed: Kirk's Current Veterinary Therapy XIII. Philadelphia: W.B. Saunders Co., 1999, p. 1004.

  • Smith, GK and McKelvie, PJ. Current concepts in the diagnosis of hip dysplasia. In: Bonagura, JD, ed: Kirk's Current Veterinary Therapy XII. Philadelphia: W.B. Saunders Co., 1995, p. 1180.

  • Teara JA, Krook L, Kallfelz FA, Hintz HF. Ascorbic acid deficiency and hypertrophic osteodystrophy in the dog: a rebuttal. Cornell Vet, 69:384-401, 1979.

  •  Wendy C. Brooks, DVM, DipABVP

  •  

  • And many others. 


Prager

by Prager on 29 June 2014 - 17:06

Blitzen  you are right....I am sorry.  I am done on this topic I said all i want to say. 

 Prager Hans






 


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